The Role of Cathepsin X in Infection With the Helicobacter Pylori

The recruitment status of this study is unknown because the information has not been verified recently.
Verified March 2010 by University Medical Centre Ljubljana.
Recruitment status was  Recruiting
Sponsor:
Information provided by:
University Medical Centre Ljubljana
ClinicalTrials.gov Identifier:
NCT01137942
First received: June 2, 2010
Last updated: July 9, 2010
Last verified: March 2010

June 2, 2010
July 9, 2010
November 2008
December 2009   (final data collection date for primary outcome measure)
Evidence that cathepsin X influences on the eradication of Helicobacter pylori confirmed by lower expression of cathepsin X and cytokines in those patients. that can not eradicate Helicobacter pylori. [ Time Frame: 7 months after last participant included in the study ] [ Designated as safety issue: No ]
The investigators assume that vast majority of patients, that have problems with eradication of Helicobater pylori, not caused by primary resistence to antibiotics, can not eradicate helicobacter because of inappropriate immune response. The investigators will measure cathepsin X (CTSX) expression and assume that those patients who have low concentrations of CTSX also have inappropriate immune response seen in low levels of cytokines. To treat such patients, you need to give them different and longer antibiotic therapy.
Same as current
Complete list of historical versions of study NCT01137942 on ClinicalTrials.gov Archive Site
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The Role of Cathepsin X in Infection With the Helicobacter Pylori
Helicobacter Pylori and Gastric Cancer - the Role of Cytokine Polymorphism, Cytokine Expression and Expression of TLR on Persistence of Helicobacter Pylori Infection and Development of Gastric Cancer.

The immune response to Helicobacter pylori (Hp) importantly determines the pathogenesis of infection as well as the success of antibiotic eradication of the bacteria. The investigators want to demonstrate the importance of cathepsin X (CTSX), a cysteine protease, for the Hp eradication success. The diversity of the innate immune response to H. pylori antigens leading to either successful eradication of the infection or maintenance of chronic inflammation is connected to CTSX. The aim of this study is to determine whether H. pylori suppresses the CTSX expression and cytokine secretion in macrophage cell line THP-1 in the individuals that are not capable of eradicating the infection, opposite to H pylori in patients with successful H pylori eradication . The investigators also investigate the possibility whether strain-dependent differences in H. pylori lipopolysaccharide (LPS) influence the CTSX expression and cytokine secretion.

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Observational
Observational Model: Case Control
Time Perspective: Prospective
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Retention:   Samples With DNA
Description:

Helicobacter pylori strains from patients are stored at -70oC.

Non-Probability Sample

The investigators invited people who had problems with H. pylori(Hp) infection. They were tested for Hp and if positive they were enrolled in the study.All patients were treated with appropriate therapy.3 months after antibiotic therapy, the patients were re-examined. The patients that had a positive test were invited to another re-evaluation.If H. pylori sensitive to all antibiotics tested was isolated, we enrolled the patient in the study-7 patients. The investigators took the patient's first isolate and used it to prepare antigens for the study. All patients in the control group, 7 patients, were successfully cured with first attempt of antimicrobial therapy.Seven months after last patient enrolled, all the participants will be re-evaluated to see if they are still infected with Hp.

Persistence of Infection With Helicobacter Pylori
Drug: clarithromycin, metronidazole, proton pump inhibitor
appropriate dose of antibiotics and proton pump inhibitor
Other Name: No other names
H. pylori eradication failure
Those who eradicated Helicobacter pylori with appropriate antibiotic therapy and those who did not.
Intervention: Drug: clarithromycin, metronidazole, proton pump inhibitor
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*   Includes publications given by the data provider as well as publications identified by ClinicalTrials.gov Identifier (NCT Number) in Medline.
 
Recruiting
14
July 2010
December 2009   (final data collection date for primary outcome measure)

Inclusion Criteria:People with helicobacter gastritis and Helicobacter sensitive to antibiotic therapy but failure of therapy -

Exclusion Criteria:People with helicobacter gastritis who did not eradicate Helicobacter pylori because of primary resistance to antibiotics.

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Both
18 Years to 65 Years
No
Contact: Alojz Ihan, MD, PhD 0038615437493 alojz.ihan@mf.uni-lj.si
Contact: Miha Skvarc, MD 0038615437484 miha.skvarc@mf.uni-lj.si
Slovenia
 
NCT01137942
IMI2010-1, 1000-05-310123
Yes
Srecko Koren, Institute of microbiology and immunology, Medical faculty Ljubljana, Slovenia
University Medical Centre Ljubljana
Not Provided
Study Director: Alojz Ihan, MD, PhD Institute of microbiology and immunology, Ljubljana, Slovenia
University Medical Centre Ljubljana
March 2010

ICMJE     Data element required by the International Committee of Medical Journal Editors and the World Health Organization ICTRP